Is green tea beneficial for reducing high creatinine level

Green tea has many medicinal benefits including reducing the risk of strokes, reducing the risk of cardiovascular disease, lowering the risk of type II diabetes and reducing cholesterol, etc. Well, is green tea beneficial for reducing high creatinine level?

Green tea is allowed with kidney disease if you take it properly.

1. Relieve ischemia and reduce kidney damage

When kidneys are damaged, blood flow through kidney will be less due to stroke or blood clots. In this condition, kidneys will be in a ischemia-anoxia state. Some researches have shown that drinking green tea in moderation can relieve renal ischemia and reduce kidney damage.

2. Prevent the buildup of glycogen

Glycogen is the rich source of glucose. If there is much glucose, you will be at a risk of developing diabetic nephropathy. Green tea can help prevent the accumulation of glycogen in the kidneys and reduce the risk of diabetic nephropathy.

3. Reduce your risk of cardiovascular disease

Most kidney patients die from cardiovascular disease than kidney disease itself. Thus, it is very important for kidney patients to have a healthy heart. Green tea has been shown to lower total and LDL cholesterol, as well as protect the LDL particles from oxidation. Studies have shown that green tea drinker have a lower risk of cardiovascular disease.

From the above, we can see green tea is beneficial for kidney condition. Although green tea can not lower high creatinine level directly, it can reduce rising creatinine level indirectly.

Of course, it is impossible for you to reduce elevated creatinine radically only with green tea. High creatinine level is from damaged kidneys which can not excrete excess creatinine out. Thus, creatinine accumulates in blood, causing elevated creatinine level. Thus, the radical treatment for high creatinine level should repair injured kidney tissues and improve renal function. To achieve this purpose, you should take a systematic Chinese medicine treatment including Medicated Bath, Medicated Foot Bath, Micro-Chinese Medicine Osmotherapy, Medicinal Soup, Steaming Therapy and Moxibustion Therapy, etc. They can restore kidney damage and improve renal function through anti-inflammation, anti-coagulation, degradation of extracellular matrix, nutrient supplement and dilation of blood vessels. When renal function is improved, high creatinine level will decrease radically.

Email:kdtinchina@yahoo.com

Online Doctor:008615931093124

Read More

What is best medicine for lowering your creatinne level 10.9

What is best medicine for lowering your creatinne level 10.9 ? Elevated creatinine level is an indicator of kidney damage. It shows your kidneys are unable to discharge excess creatinine out of your body.

The best medicine for high creatinine level 10.9 in our hospital is Chinese medicine. We have a series of Chinese medicine therapies to lower down elevated creatinine level. For example:

Micro-Chinese Medicine Osmotherapy

This therapy is used externally based on Traditional Chinese Medicine (TCM) and Chinese medicines are micronized into powder. Processed Chinese medicines will be put into two medicated bags. Patients just need to lie on bed to take this therapy. It is just like a massage and feels very comfortable. The osmosis machine helps the active ingredients arrive at kidney lesion directly. Through dilating blood vessels, improving blood circulation, preventing inflammation and coagulation, degrading extracellular matrix and providing nutrients, it is able to restore injured kidney tissues and improve renal function. When renal function is improved, your kidneys can lower down high creatinine level 9 effectively and radically.

Moxibustion Therapy

With Moxibustion Therapy, the main channels of your body will be cleaned. The blood flow and circulation will be increased. Also your immunity will be improved. Then you will have great resistance to fight against diseases. Moxibustion therapy can help improve your sleep quality, increase urine volume, lower down high creatinine level and relieve weakness.

Medicated Foot Bath

There are acupoints in your feet which are connected with your kidneys. With Medicated Foot Bath, Chinese medicines can be transported to your kidneys directly and kidney disease can be treated by dilating your blood vessels, improving blood circulation, providing nutrients. Your condition can be improved greatly with Medicated Foot Bath.

Email:kdtinchina@yahoo.com

Online Doctor:008615931093124

Read More

So i want to know how to increase urine output in uremia

I am a uremia patient and dialysis for 5 years, i have skin itching and take western medicines to control my blood pressure. I almost have no urine in recent days, So i want to know how to increase urine output in uremia.

Most of patients choose dialysis to maintain life, as we all know it can only control the complications and illness conditions, but with no effects ob treating kidney disease. Kidney will be gradually damaged seriously, so there are lots of patients with no urine in a time. So we need try our best to control the illness conditions and improving kidney function to increase urine output.

If blood can flow into kidney that means there is kidney working. So we need to remove the toxins and excess wastes out, which will help expend blood vessels, improving blood circulation that the oxygen and nutrients will be transported to cells and tissues. The diseased cells and diseased tissues will be repaired, so kidney function will gradually improve.

Micro-Chinese Medicine Osmotherapy is an external application with two bags. Two bags fulfill with chinese herbal medicines powder. The experts will select the chinese herbal medicines for you that depends on your illness conditions. All the chinese herbal medicines used in Micro-Chinese Medicine Osmotherapy come from nature, so patients do not worry about the side effects. Doctors will put the two bags on your shenshu area and contact osmotic machine, which will help the active ingredients to be absorbed effectively. And the chinese herbal medicines will play its effects to improve kidney function.

After several times treatments that patients’ illness conditions can change a lot, the urine will be gradually increased, and the toxins and excess wastes will be cleared away, which will creative a clean and healthy internal environment for uremia patients.

Email:kdtinchina@yahoo.com

Online Doctor:008615931093124

Read More

2017 five areas of kidney disease progress, do you know a few

Looking back at 2017, the field of kidney disease is also very profitable and has made many progress. Recently, Nature magazine gave a detailed overview of 5 major advances in kidney disease in 2017. Have you ever missed a chance?

Dr.Leo

https://www.facebook.com/LifeforKidneypatients/

1.A new mechanism of hypertension and vascular dysfunction

In 2017, a number of basic and clinical studies published in the field of hypertension further deepen our understanding of the mechanism of blood pressure regulation. New discoveries include: immune mechanisms, the role of gut microbes, the adverse effects of perivascular fat and inflammation on the vascular system, and the effects of rare mutations in the renin-angiotensin-aldosterone system on salt sensitivity.

Important progress:

➤ The SGK1 signaling pathway in T cells upregulates Na + / K + / 2Cl-co-transporter 1 (NKCC1) and promotes phenotypic polarization of helper T-cell 17 (TH17) cells, resulting in Lead to salt-sensitive hypertension and end-stage ductal injury.

➤ A high-sodium diet alters the gut microflora in humans and mice, resulting in the depletion of lactobacilli, increasing the number of TH17 cells and increasing blood pressure.

➤ γδ T cells lead to angiotensin II-induced hypertension and endothelial dysfunction in mice and may also play important roles in human hypertension and end organ damage.

➤ AGTR1A and osteopontin are involved in the polarization of phenotypic macrophages in perivascular fat-inflammation, leading to inflammation and promoting aortic aneurysm formation.

➤ Rare mutations in the renin-angiotensin-aldosterone system (RAAS) -related gene (7 in total, including APLN and RENBP) are associated with salt-sensitivity to blood pressure.

Hypertension and aortic aneurysm form new mechanisms

2. glycolysis regulation, promote kidney disease progress

There is growing evidence that cellular metabolism is related to the fate of cells. In particular, glycolytic fluxes have become recognized as important drivers of self-renewal and cell proliferation and are crucial for angiogenesis and tumor growth. Several studies published in 2017 highlight the important role of glycolysis and mitochondrial function in kidney development and kidney function, as well as the impact on kidney disease progression.

Important progress:

➤ Increased glycolysis and mitochondrial respiratory function promote progenitor cell proliferation and self-renewal.

➤ Acute kidney injury and diabetic nephropathy are characterized by mitochondrial dysfunction and increased glycolytic intermediates - which can be metabolized to toxic metabolic end products.

➤ Coupling of glycolysis flux to mitochondrial respiratory function is becoming a potential treatment for ischemic and diabetic kidney damage.

Glycolytic metabolism; PFKFB3: 6-phosphofructo-2-kinase / fructose-2,6-bisphosphatase 3; AMPK: AMP-activated protein kinase; PKM2: pyruvate kinase M2; LDH: lactate dehydrogenase; -6-P: glucose 6-phosphate; PGC-1α: Peroxisome Proliferator Receptor γ Coactivator 1α

3. Immune nephropathy mechanism and progress of treatment

In the past year, the research on the pathogenesis and treatment of immune-mediated glomerular diseases has been deepened and many progresses have been made. In the field of molecular transformation, two studies have found new pathogenesis and pathways, providing new treatments for the treatment of immune nephropathy - hormones may or may not be used in the future.

Important progress:

➤ The reason that HLA is associated with anti-glomerular basement membrane disease is that immunodominant epitope peptides bind to different HLA.

➤ Autoantigen DNA Methylation - Induced Antineutrophil Cytoplasmic Antibody (ANCA) -associated Vasculitis - May Be Associated with Disease Activity and Remission, and Predict Disease Activity and Remission.

➤ High-dose glucocorticoid treatment for IgA nephropathy has more side effects than potential benefits.

➤ Targeted Targeted Release Agent (TRF) Targeting Intestinal Immune Tissue - Budesonide is effective in treating IgA nephropathy and significantly reduces hormone use

➤ Small molecule C5a receptor inhibitors improve patient outcomes, meaning that complement plays an important role in ANCA-associated glomerulonephritis. Small molecule C5a receptor inhibitors are as effective as prednisone in inducing disease remission

4. Diabetic nephropathy treatment of a new era

Since 16 years ago IDNT and RENAAL studies have found no new drugs approved for use in patients with type 2 diabetes (T2DM) and kidney disease using the angiotensin II receptor blocker irbesartan and losartan Treatment of diabetic nephropathy. In 2017, however, studies have shown that the hypoglycemic agents sodium-glucose cotransporter 2 (SGLT2) inhibitors (enzaprine, canagliflozin) and glucagon-like peptide-1 (GLP- Liraglutide) has a protective effect on the kidney, bringing new hope for patients with diabetic nephropathy.

Important progress:

➤ CANVAS and EMPA-REG OUTCOME studies have reported that sodium-glucose cotransporter 2 (SGLT2) inhibitors are beneficial for renal outcomes, including proteinuria, and may reduce creatinine levels and reduce renal replacement therapy or kidney death.

➤ LEADER study shows that the GLP1 agonist liraglutide can reduce the incidence of diabetic nephropathy and delay the progression of the disease.

➤ Previous renal disease, estimated glomerular filtration rate of 30-60 ml / min / 1.73 m2 and / or large amounts of proteinuria, the use of enzapine can significantly reduce the risk of cardiovascular death, all-cause mortality risk and Heart failure hospitalization rate and all-cause hospitalization rate.

Effects of SGLT2 inhibitors and GLP1 agonists on diabetic renal hemodynamics; a: Hemodynamic changes due to hyperglycemia and diabetes; b: Effects of SGLT2 inhibitors and GLP1 agonists on hemodynamics; JGA: Golotrophore; NHE3: sodium-hydrogen exchanger 3; RAS: renin-angiotensin system; SNS: sympathetic nervous system

5. Uncover the mystery of the genetic structure of kidney disease

With the continuous development of gene sequencing technology, we have made a major breakthrough in understanding the genetic structure of kidney disease. In 2017, four major studies reveal the genetic basis of known diseases and novel diseases, providing new insight into the pathogenesis of glomerular diseases, kidney defects and clinical manifestations.

Important progress:

➤ SGPL1 recessive mutations lead to abnormal sphingolipid metabolism and are associated with steroid-resistant nephrotic syndrome.

Repeated loss of the 22q11.2 locus leads to DiGeorge syndrome, congenital kidney and urethral deformities; single-dose CRKL may also play a role.

➤ An unchallenged family history of autosomal dominant polycystic kidney disease has been a major clinical diagnostic difficulty, probably due to de novo lesions, germ cells or somatic chimeras, or mild disease due to PKD1 or PKD2 secondary morphological changes .

➤ Unidentified genomic diseases may impair the development of children's kidneys and neurocognitive development, and early detection can provide immediate intervention.

 A new pathway of congenital nephropathy; a: role of SGPL1 in the sphingolipid pathway; b: adapter protein CRKL pathway; CDase: ceramidase; CS, ceramide synthase

Email:kdtinchina@yahoo.com

Online doctor:008615931093124

Read More

The 13th World Kidney Day 2018 coincides with the International Women's Day

The 13th World Kidney Day 2018 coincides with the International Women's Day, whose theme is "Concerned Kidney Disease and Caring for Women's Health" and provides us with a focus on the health of women, especially the health of women's kidneys.

The prevalence of glomerular disease has a gender difference, and a global histological diagnostic survey shows that the prevalence of glomerular disease in women is 47% and in men 53%.

Female glomerular disease medication

The treatment of glomerular diseases includes immunosuppressive agents, angiotensin receptor blockers, antihypertensive drugs, and management of CKD-related complications. Glomerular disease women should be fully discussed before pregnancy.

Immunosuppressant

Women taking teratogenic immunosuppressive agents (mycophenolate mofetil, methotrexate, and cyclophosphamide) should take active contraception in a safe and effective way of contraception. Women who plan to become pregnant as well as women who may have unwanted pregnancies should avoid the use of these drugs. Alternative immunosuppressants that are safe to use during pregnancy are glucocorticoids, calcineurin inhibitors, azathioprine and hydroxychloroquine. Cyclophosphamide, mycophenolate mofetil, leflunomide and methotrexate have teratogenic effects, contraindication during pregnancy should be at least 3 to 6 months before conception disabled.

Angiotensin receptor blockers and antihypertensive drugs

The use of angiotensin receptor blockers in the second and third trimester of pregnancy is toxic to the fetus and can result in oligohydramnios and neonatal renal failure. Used during early pregnancy, no increase in the incidence of congenital abnormalities. Therefore, glomerular disease in women can continue to use before the diagnosis of pregnancy. Antihypertensive drugs that are safe during pregnancy include methyldopa, labetalol and long-acting nifedipine. The safety of amlodipine is not well understood, but case reports and small studies suggest the drug is not harmful.

Other drugs

There are many reasons for anemia in glomerular diseases, including the worsening of iron deficiency due to menstrual blood loss and the reduction of erythropoietin in CKD. Pregnant women can safely use oral and intravenous iron. During pregnancy can continue to use low-dose aspirin for vascular disease, primary and secondary prevention.

Finally, send a most sincere blessing to women around the world.

Dr.Leo

https://www.facebook.com/LifeforKidneypatients/

Read More