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Potassium is an important material to maintain cardiac function, hypokalemia easily lead to arrhythmia. When the extracellular or blood potassium concentration decreased, the difference between the potassium concentration inside and outside the cell membrane increased, the membrane potassium permeability decreased. Hypokalemia, increased cardiac excitability, increased self-discipline, reduced conductivity.
When the difference of potassium concentration inside and outside of myocardial cells increased, the potassium permeability of the membrane decreased and the outflow of potassium ions decreased, which decreased the resting potential of cardiomyocytes. As the membrane potential decreases close to the threshold potential, increased cardiac myocyte excitability. At the same time, the effect of relative influx of sodium ions is weakened, the speed of automatic depolarization of diastolic myocardial cells is accelerated, the time from the diastolic potential after repolarization reaches the threshold potential is shortened, and the self-discipline of cardiomyocytes is increased. And due to reduced resting potential, sodium influx and depolarization speed and amplitude decreased, so slow conduction of excitement.
Thus, due to hypokalemia due to changes in cardiomyocytes Na + and K + channel permeability, increased cardiac excitability, increased self-discipline, reduced conductivity, easy to form excited reentry, and induced arrhythmia.
Hypokalemia is the early symptoms of limb weakness, generalized weakness, bloating, nausea, constipation, lack of energy and other symptoms, and mild hypokalemia caused by arrhythmia mostly sinus tachycardia, atrial contraction Or ventricular contractions before.
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